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S100A1 be part of a major group consisting of proteins be used S 100 will high levels in high concentrations in the muscular , particularly the heart. Falling levels of S100A1 has are crucial for loss from heart-pumping strength of following a heart attack and have an important role in the progression to heart failure. An earlier study the 1989 indicated that the protein by more than 50 % for patients with heart failure. 29.2 % The current work, Most and colleague Andrea Eckhart, associate professor of medicine from the Jefferson Medical College, and her team are in laboratory testing and animal models that blood vessels missing S100A1 unable to relax as well normal vasculature. ‘When the animal does not S100A1 has is was hypertension,’he says. ‘This mechanism is a greater or lesser is based to availability of the nitrous oxide. And it seems that S100A1 also provides for of calcium Cycling in endothelial cell, and calcium in the endothelial cell is will be required the NO production the production of NO. Loss of S100A1 affects calcium mobilizing mobilizes endothelial cells of – connection between the connection between less of calcium, less NO , hypertension and endothelial function ‘As an result,’cider says, ‘S100A1 has would is not only have a good therapeutic objective of heart failure, but for hypertension. Well as ‘latest forecasts appreciate that 29.2 % of the adult population the world – from about 1, Moreover human – hypertensive Hypertension 2025 has long been a most common risk factor for development of heart failure and affects almost five million Americans. Many of them poor long -term prognoses, spite advances in treatment.

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